Azithromycin or ciprofloxacin for diarrhea -predominant strains of E. coli.
"The problem is not with these antibiotics," says Dr. Peter Rabe, a microbiologist at Massachusetts General Hospital in Boston. "The problem is a breakdown in the microbiome. And a breakdown in the microbiome has been correlated with obesity and metabolic syndrome. We're trying to find out how the microbiome changes in patients who have been taking these antibiotics long term. This could be the first step toward changing that microbiome."
Another promising strategy involves a vaccine that targets the microbiome itself. Last year, scientists reported in Nature that administering the DNA of an E. coli bacteria into the intestines of mice could reprogram the organisms to a more beneficial state—one that, in part by preventing the production of bacterium's own antibiotic-degrading enzymes, would make the animal less susceptible to antibiotic-resistant infections. By the same token, it would produce a less resistant bacterial culture, which would reduce the need to keep such bacteria alive.
That approach might also be applicable to the human colon—a potentially better way of controlling the pathogenic bacterial species that inhabit it. "You could imagine a vaccine that was targeted to specific species, like Enterococcus," Srivastava says, which would make it useful not just for infections but also treating gastrointestinal diseases such as ulcerative colitis. "That's certainly exciting."
Srivastava also notes that the potential for such approaches Tamsulosin price australia is already apparent in the field of nutrition: buy azithromycin online canada "The same microbiome that determines how we metabolize food could also contribute to how our gut microbiome controls we eat," he says. In a recent review Nature Reviews, the microbiologist Mark Davis of Johns Hopkins University School Medicine called these emerging research questions "the next frontier of microbiome research."
For his part, Srivastava has started to take such studies more seriously. A graduate student last September, he has been conducting a large study of the microbial community in two of the world's most contaminated sites—the Los Angeles and Tokyo subway systems. In the past decade, many microbiologists and nutritionists have been alarmed by an alarming increase in obesity and diabetes rates the United States, European Union, and Japan, as well a dramatic decline in physical activity. "We see the same issues here," Srivastava says. "We're not doing an exhaustive analysis yet, but we're starting to see this relationship."
It's difficult to know which of these theories will be the most fruitful to pursue, which ones will lead to better therapies for diabetes and obesity, or which combination of factors will ultimately result in optimal health. "It's a complex issue," Srivastava says. "There's probably an unknown number of mechanisms that interact to help explain the link. We have basic science now; it's time to test."
Savin Shukla is a science journalist and advocate. He has worked as a public editor at Science, has written for The Los Angeles Times Book Review, and was a contributing editor at Discover.
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Azithromycin or ciprofloxacin for diarrhea -predominant strains of E. coli. "The problem is not with these antibiotics," says Dr. Peter Rabe, a microbiologist at Massachusetts General Hospital in Boston. "The problem is a breakdown in the microbiome. And a breakdown in the microbiome has been correlated with obesity and metabolic syndrome. We're trying to find out how the microbiome changes in patients who have been taking these antibiotics long term. This could be the first step toward changing that microbiome." Another promising strategy involves a vaccine that targets the microbiome itself. Last year, scientists reported in Nature that administering the DNA of an E. coli bacteria into the intestines of mice could reprogram the organisms to a more beneficial state—one that, in part by preventing the production of bacterium's own antibiotic-degrading enzymes, would make the animal less susceptible to antibiotic-resistant infections. By the same token, it would produce a less resistant bacterial culture, which would reduce the need to keep such bacteria alive. That approach might also be applicable to the human colon—a potentially better way of controlling the pathogenic bacterial species that inhabit it. "You could imagine a vaccine that was targeted to specific species, like Enterococcus," Srivastava says, which would make it useful not just for infections but also treating gastrointestinal diseases such as ulcerative colitis. "That's certainly exciting." Srivastava also notes that the potential for such approaches Tamsulosin price australia is already apparent in the field of nutrition: buy azithromycin online canada "The same microbiome that determines how we metabolize food could also contribute to how our gut microbiome controls we eat," he says. In a recent review Nature Reviews, the microbiologist Mark Davis of Johns Hopkins University School Medicine called these emerging research questions "the next frontier of microbiome research." For his part, Srivastava has started to take such studies more seriously. A graduate student last September, he has been conducting a large study of the microbial community in two of the world's most contaminated sites—the Los Angeles and Tokyo subway systems. In the past decade, many microbiologists and nutritionists have been alarmed by an alarming increase in obesity and diabetes rates the United States, European Union, and Japan, as well a dramatic decline in physical activity. "We see the same issues here," Srivastava says. "We're not doing an exhaustive analysis yet, but we're starting to see this relationship." It's difficult to know which of these theories will be the most fruitful to pursue, which ones will lead to better therapies for diabetes and obesity, or which combination of factors will ultimately result in optimal health. "It's a complex issue," Srivastava says. "There's probably an unknown number of mechanisms that interact to help explain the link. We have basic science now; it's time to test." Savin Shukla is a science journalist and advocate. He has worked as a public editor at Science, has written for The Los Angeles Times Book Review, and was a contributing editor at Discover.
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